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Re: Alzheimer's Disease
[Re: Suzanne]
#147589
11/27/12 07:18 PM
11/27/12 07:18 PM
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A workout a day keeps dementia away
by Ben Meredith
(NaturalNews) It's common knowledge that exercise is good for the body. Regular exercise aids in reducing the risk of heart attack and stroke, among other important benefits. New research now shows, though, that exercise can prove to be a very vital factor in the lessening or prevention of cognitive impairment.
Doctors at the University of Lisbon in Portugal organized a study of over 600 participants between the ages of 65 to 84. Each of the participants' brains showed early warning signs of dementia, but none of them had debilitation in their daily lives, and they all lived independently. The researchers followed the subjects over a period of three years. During that time, the participants had their brain scanned on a regular basis. They were assessed on their cognitive ability, and they reported how often and how much they exercised.
The results showed that the participants who reported doing 30 minutes of exercise at least three times per week had about a 40 percent lower risk of developing dementia in comparison to those who reported less activity. During the three years of the study, 90 subjects developed dementia.
"Dementia" is a term that describes multiple types of mental disability. The most common mental disability is Alzheimer's. In this study, the exercise-to-cognitive-function association was strongest for vascular dementia, which is a type of non-Alzheimer's dementia that is caused by inadequate blood flow to the brain. While exercise showed a reduction in the risk for vascular dementia by almost 60 percent, physical activity didn't seem to specifically affect the risk of developing Alzheimer's.
The doctors performing the study also took MRI scans of their subjects to look for changes in the white matter of the participants' brains, which is linked to dementia. All of the participants showed changes of white matter at the beginning of the study in varying degrees. Researchers made sure to take into account other factors that can affect memory and cognitive function, like age, history of stroke, and diabetes. However, they found that even in taking in these other factors, the protective effects of exercise still held.
There were 34 cases in the study whose findings didn't hold. These participants showed signs of Alzheimer's, and in their cases, exercise was not correlated to their rates of mental decline.
It was noted by the professionals that, before starting an exercise program, patients should discuss their plans and get clearance from their doctors. A new exercise program should begin in a gradual manner, especially for those with heart problems. But even if physical activity doesn't delay mental decline, it's still a win-win, as it has many other important health benefits that shouldn't be overlooked.
Sources for this article include:
http://www.medpagetoday.com/Cardiology/Dementia/35685
http://www.cbc.ca
http://www.counselheal.com
Suzanne
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Re: Alzheimer's Disease
[Re: Suzanne]
#160304
01/14/14 12:35 AM
01/14/14 12:35 AM
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Want to save your brain from dementia? Hit the treadmill and pump some weights
by David Gutierrez, staff writer
(NaturalNews) The longest-ever study examining the effect of lifestyle choices on chronic disease has concluded that moderate exercise may be the single most effective way to prevent dementia.
"What the research shows is that following a healthy lifestyle confers surprisingly large benefits to health," lead researcher Peter Elwood said. "Healthy behaviors have a far more beneficial effect than any medical treatment or preventative procedure."
The study, conducted by researcher from the Cardiff University and published in the journal PLOS ONE, followed 2,235 men from south Wales for 35 years, starting in 1979. The men, aged 45 to 49 at the study's start, answered regular questions about their lifestyle and health throughout the study.
The researchers found that five factors were most important in reducing rates of all chronic diseases, including dementia: regular exercise, healthy diet, low body weight, low alcohol intake and not smoking. Compared with people who followed none of these five factors, people who followed any four were 60 percent less likely to suffer from dementia or cognitive decline and 70 percent less likely to suffer from diabetes, heart disease or stroke.
"The size of reduction in the instance of disease owing to these simple healthy steps has really amazed us and is of enormous importance in an ageing population," Elwood said.
Improve your health today
The single biggest connection found was that between exercise and dementia, yet Elwood emphasized that an overall healthy lifestyle is more important than any one component.
"Exercise happens to be the most important but the other factors come in very close behind," he said.
Yet, increasing your physical activity level might be one of the easiest ways to improve your overall health. It takes only 30 minutes of exercise five days a week to reap significant benefits.
"We should all live a more active lifestyle," Elwood said. "If I park my car a mile from work - that makes me likely to do more than the half an hour a day. Any exercise has some benefit and the more, the better."
"Sadly, the evidence from this study shows that very few people follow a fully healthy lifestyle," Elwood said.
The researchers found that, even with reductions in overall smoking rates over the past 35 years, the number of people who qualified as living a healthy lifestyle did not change during the course of the study. This is consistent with recent surveys finding that less than 1 percent of the population of Wales follows all five recommended factors, with 5 percent not following any.
"If the men [in the study] had been urged to adopt just one additional healthy behavior at the start of the study 35 years ago, and if only half of them complied, then during the ensuing 35 years there would have been a 13 percent reduction in dementia, a 12 percent drop in diabetes, 6 percent less vascular disease and a 5 percent reduction in deaths," Elwood said.
The researchers also found that, over the course of the study, unhealthy living was responsible for 10 percent of all healthcare expenditure in Wales.
Dementia is a growing public health concern, expected to affect 135 million people worldwide by 2050.
"We are facing potentially large increases in the numbers of people living with dementia and if we are to deal with this crisis head on we must invest in research," said Rebecca Wood, chief executive of Alzheimer's Research UK.
"It's encouraging for people to know there are simple steps they can take now to reduce their risk," she said.
Sources for this article include:
http://www.bbc.co.uk
http://www.belfasttelegraph.co.uk
http://www.express.co.uk
http://science.naturalnews.com
Suzanne
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Re: Alzheimer's Disease
[Re: Suzanne]
#161265
02/04/14 12:14 AM
02/04/14 12:14 AM
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Exposure to DDT linked to increased risk of Alzheimer's disease in elderly adults
by L.J. Devon, Staff Writer
(NaturalNews) Even after being banned more than 40 years ago, the pesticide DDT continues to affect the lives of people today, beckoning infertility and other deeply concerning health conditions. Researchers are now finding out how the pesticide affects the nervous system. In 1972, when the US banned DDT from commercial production, environmental tests confirmed that this pesticide was harmful to the environment, especially bird habitats.
Now, decades later, this toxic pesticide is being linked to increased risk of Alzheimer's in elderly adults. Researchers at Rutgers University report that DDT increases risk and severity of Alzheimer's disease in patients primarily over the age of 60. The findings, published in the journal JAMA Neurology, showed how DDE, the breakdown compound of DDT, is measured four times higher in the blood of late onset Alzheimer's patients. The study took into consideration only chlorinated compounds and did not assess newer pesticides widely accepted in agriculture today.
DDT lurking in most blood samples today
Introduced as a pesticide during WWII, DDT was applied en masse as agricultural insect control and was even used to fight off insect-borne diseases like malaria. (Africa still uses this pesticide in homes to control insects.) The pesticide is so pervasive that it regularly appears in 75 to 80 percent of the blood samples collected today, according to the Centers for Disease Control.
This may be due to imported fruit that comes from countries that still apply DDT. This may also be because DDT takes many decades to break down in the environment. Traces of WWII-era DDT could be breaking down in people's blood today in the 21st century.
DDT levels measured four times greater in Alzheimer's patients
For the Rutger's study, the Emory University Alzheimer's Disease Research Center and the University of Texas Southwestern Medical School's Alzheimer's Disease Center collaborated, focusing on 86 Alzheimer's patients and 79 control group participants who showed no symptoms of Alzheimer's. When the researchers measured the blood of all participants, they found that 74 out of 86 Alzheimer's patients had DDE blood levels four times greater than control participants!
Their study connected genetics and environmental factors. Alzheimer's patients showing expression of the gene ApoE typically possess greater risk for Alzheimer's; in concordance, the highest measured levels of DDE were correlated with the most severe cognitive impairments in those with ApoE gene expression.
"We need to conduct further research to determine whether this occurs and how the chemical compound interacts with the ApoE4 gene," says Jason R. Richardson, associate professor in the Department of Environmental and Occupational Medicine at Robert Wood Johnson Medical School.
DDT pesticide increases nerve cell plaque associated with Alzheimer's
Alzheimer's is characterized by plaque formation between nerve cells, which leads to massive nerve cell die-offs. Increased amyloid protein permits the plaque to form.
In further brain cell studies, the researchers made an association between pesticide DDT and increased amyloid protein accumulation. DDT basically fueled nerve cell plaques in areas of the brain responsible for learning, memory and thinking.
"Much of the research into Alzheimer's and other neurodegenerative diseases has mostly been centered on finding genetic connections," Richardson says. "I think these results demonstrate that more attention should be focused on potential environmental contributors and their interaction with genetic susceptibility."
"This study demonstrates that there are additional contributors to Alzheimer's disease that must be examined and that may help identify those at risk of developing Alzheimer's. It is important because when it comes to diagnosing and treating this and other neurodegenerative diseases, the earlier someone is diagnosed, the more options there may be available."
Sources for this article include:
http://news.rutgers.edu
http://www.environmentalhealthnews.org
http://science.naturalnews.com
Suzanne
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Re: Alzheimer's Disease
[Re: Suzanne]
#161305
02/04/14 07:13 PM
02/04/14 07:13 PM
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Natural Flavonol Compound in Fruits and Vegetables Prevents Alzheimer's Disease
by L.J. Devon, Staff Writer
(NaturalNews) Riddled with deposits of a peptide called beta-amyloid, the brain can become consumed with plaque, which builds up in the spaces between nerve cells. When nerve cells begin to die off, symptoms of Alzheimer's disease set in.
Natural flavanol works beyond plaque to restore memory
In areas of the brain where memory is important, tangles of plaque can develop from twisted fibers of tau protein. Alzheimer's, the most common form of dementia, has always been recognized by this plaque buildup, which is associated with problems in thinking, memory and behavior. As a condition that slowly worsens over time, Alzheimer's is ultimately capable of interfering with daily tasks and newly learned information.
New findings suggest that Alzheimer's can be reversed. A new fruit- and vegetable-based treatment could effectively bypass plaque formation and work independently to restore memory in the nerve cells of the brain, turning on specific memory-related pathways.
Nerve cells work together in vast networks
With over 100 billion nerve cells at work in the brain, communicating in vast networks, cellular protection is vital. Nerve cells specialize in some of the most complicated areas of the human experience, including jobs like smell, taste, hearing, learning, thinking,and memory. Operating like miniature factories, nerve cells perform an array of functions, including obtaining necessary supplies, communication, energy generation, information storage and waste removal. Scientists have a hard time pinpointing how Alzheimer's takes hold in a person as they age.
Fisetin improves memory of Alzheimer's-ridden mice
How might specific properties of fruits and vegetables help stop memory loss as seen in Alzheimer's disease?
One flavanol, fisetin, was isolated, studied and put to the test in Alzheimer's-ridden mice. Scientists at the Salk Institute for Biological Studies have discovered that a daily dose of this fruit and vegetable compound can help mice recover their memory.
The fisetin surprised the scientists, showing promise for improving memory even as amyloid plaque formation stayed the same. These accumulations of proteins remained prevalent in Alzheimer's mice and continued gumming up nerve cells even after fisetin was administered, but their memory improved independent of the plaque formations. This new finding suggests that there may be a way to treat Alzheimer's symptoms without combating amyloid plaques.
"Fisetin didn't affect the plaques," says Maher. "It seems to act on other pathways that haven't been seriously investigated in the past as therapeutic targets," says Pamela Maher, a senior staff scientist in Salk's Cellular Neurobiology Laboratory who led the new study.
Over 10 years ago, Maher learned that fisetin helped protect neurons in the brain, after isolating the flavanol in cell cultures and examining its anti-inflammatory, antioxidant prowess in nerve cells. In her research, fisetin was found to turn on a specific cellular pathway that improves memory.
Fisetin working on the molecular level
Working with Dave Schubert, leader of the Cellular Neurobiology Lab, Maher conducted her tests on a group of mice with two gene mutations linked to Alzheimer's.
After feeding the fisetin to the mice at three months old, the researchers began studying how the natural substance affected mice in a variety of learning skills and water mazes.
At just nine months of age, mice receiving no fisetin performed more poorly in the water mazes. These Alzheimer's-destined mice would normally show memory deficits by the first year, but after eating fisetin, they performed just as well as normal mice, at both nine months old and one year of age.
"Even as the disease would have been progressing, the fisetin was able to continue preventing symptoms," Maher says.
To investigate further, Maher collaborated with scientists at the University of California, San Diego, to key in on specific brain molecules. What they found was that fisetin turned on pathways involved in cellular inflammation. Anti-inflammatory molecules were observed in specific areas of the brain involved in memory after pathways were triggered on.
When fisetin was administered, p35 protein was blocked from being cut down into a shorter form. Short versions of the p35 protein are responsible for turning many molecular pathways on and off.
"It may be that compounds like this that have more than one target are most effective at treating Alzheimer's disease," says Maher, "because it's a complex disease where there are a lot of things going wrong."
Sources for this article include:
http://www.salk.edu
http://science.naturalnews.com
Suzanne
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Re: Alzheimer's Disease
[Re: Suzanne]
#163683
03/25/14 06:12 PM
03/25/14 06:12 PM
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New Risk Factor For Alzheimer's Disease Revealed: Are You Getting a Good Night's Sleep?
by John Phillip
(NaturalNews) Extensive research over the past twenty years has repeatedly shown that Alzheimer's dementia is largely the result of a variety of lifestyle factors that promote the development of amyloid plaques and tau proteins in the brain that usher the onset of this dreaded disease. A diet high in processed carbohydrate and sugars, exposure to environmental and household pollutants and lack of physical activity have all been linked with development and progression of the disease. Now scientists have found that poor sleep patterns, as experienced by millions of aging adults, may be a powerful trigger for Alzheimer's disease.
Researchers from Temple University in Philadelphia, PA, reporting the results of their work in the journal, Neurobiology of Aging, have determined that people who experience chronic sleep disturbance, either through their work, insomnia or other reasons, could face an earlier onset of dementia and Alzheimer's. Prior research bodies have concluded that chronic sleep problems can inflame a number of health problems, ranging from cardiovascular disease and depression to cancer and diabetes.
Lack of restful sleep promotes brain tangle formation and early signs of Alzheimer's dementia
Lead study author, Dr. Domenico Pratico commented "The big biological question that we tried to address in this study is whether sleep disturbance is a risk factor to develop Alzheimer's or is it something that manifests with the disease." Using a transgenic mouse model known to simulate human neurological pathologies, the team examined the effects of sleep deprivation to determine the development of the two hallmark signs of Alzheimer's disease: amyloid plaques and tau protein tangles.
Starting their study when the mice were 6 months old, the human equivalent of a 40-year-old, the researchers began an 8-week study. As one group of mice was exposed to 12 hours of light and 12 hours of darkness, another group was exposed to 20 hours of light and 4 hours of darkness, which significantly reduced their sleep time. At the end of the test period, the scientists did not detect any outward behavioral signs of Alzheimer's disease. When they conducted memory tests in the mice however, the group with reduced sleep time demonstrated significant impairment in their working and retention memory, as well as the ability to learn new tasks.
Dr. Pratico noted "We did observe that the sleep disturbance group had a significant increase in the amount of tau protein that became phosphorylated and formed the tangles inside the brain's neuronal cells." Tau protein is an important part of neuronal cell health, so these elevated levels cause a disturbance in normal function. This disruption will eventually impair the brain's ability for learning, forming new memory and other cognitive functions, and contributes to Alzheimer's disease.
The authors concluded "that chronic sleep disturbance is an environmental risk factor for Alzheimer's disease... but the good news is that sleep disturbances can be easily treated, which would hopefully reduce the Alzheimer's risk." It has been well documented that we require 7 to 9 hours of restful, uninterrupted sleep in a fully darkened room each and every night to promote efficient metabolic housekeeping and ward off a number of chronic disease conditions including Alzheimer's dementia.
Sources for this article include:
http://www.neurobiologyofaging.org
http://www.medicalnewstoday.com/articles/274188.php
http://www.sciencedaily.com/releases/2014/03/140317155205.htm
Suzanne
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Re: Alzheimer's Disease
[Re: Suzanne]
#164799
05/06/14 11:49 PM
05/06/14 11:49 PM
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Exercise Halts 'Dementia Gene' From Being Expressed, Keeps Your Brain Healthy
by Jonathan Benson, staff writer
(NaturalNews) Dementia is the sixth-leading cause of death in the U.S. today, and experts say the next generation could see twice as many people suffering from it, particularly in old age. But avoiding the brain-deteriorating disease could be as simple as remembering to exercise just a few times a week, according to a new study, which appears to inhibit the expression of the "dementia gene."
Over the course of 18 months, researchers from the University of Maryland measured the brain sizes of elderly participants who were divided into four groups. The activity levels of each of the participants were gauged in conjunction with whether or not they possessed a gene known as APOE-e4, which has been associated with an increased risk of dementia.
Up to 30 percent of the population possesses this gene, and the research team wanted to see whether or not physical exertion affects its expression. What they found is that those with APOE-e4 who exercised at least three times a week experienced a lesser overall decrease in brain mass, a common sign of dementia, compared to those who engaged in little or no exercise.
"We found that physical activity has the potential to preserve the volume of the hippocampus in those with increased risk for Alzheimer's disease, which means we can possibly delay cognitive decline and the onset of dementia symptoms in these individuals," stated Dr. J. Carson Smith, one of the study's authors. "Physical activity interventions may be especially potent and important for this group."
Don't forget proper nutrition in the fight against dementia
Published in the journal Frontiers in Aging Neuroscience, the study provides new insights into the mind-body connection, and the importance of physical exertion in maintaining a healthy brain. Though exercise alone is hardly the best approach, as nutrition is equally if not more important for preserving brain function, it is vital for maintaining good health, especially in old age.
"We do not yet have the level of exercise needed to justify this approach specifically for Alzheimer's prevention," added Dr. Smith, as quoted by the UK's Daily Mail. "[B]ut exercise certainly cannot harm, so should be prescribed regardless."
To go along with this, avoiding certain foods and chemical exposures and embracing better dietary and lifestyle habits will duly help in the fight against dementia....
Sources for this article include:
http://www.dailymail.co.uk
https://www.alz.org
http://wellnessmama.com
http://www.psychologytoday.com
http://science.naturalnews.com
Suzanne
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Re: Alzheimer's Disease
[Re: Suzanne]
#164961
05/13/14 05:49 PM
05/13/14 05:49 PM
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Could The Key To Alzheimer's Disease Prevention Be As Simple As Eating Strawberries?
by John Phillip
(NaturalNews) Two decades of dedicated research has clearly revealed that dementia and specifically the dreaded diagnosis of Alzheimer's disease can be prevented or even halted by making lifestyle modifications toward a natural food diet that excludes processed and refined junk foods as well as following a regular exercise regimen and limiting exposure to personal cosmetics and environmental toxins. While this sounds like a tall order for many, slow but steady progress toward a more natural lifestyle can be beneficial to many individuals who may be headed toward a host of chronic diseases associated with aging including Alzheimer's. An extensive body of research over the past five to ten years has validated certain natural compounds found in fruits and vegetables, specifically the delicious strawberry.
Researchers from the Salk Institute for Biological Studies has found that fisetin, a flavanol found in strawberries, mangoes, cucumber and other fruits and vegetables, may protect the brain against Alzheimer's, dementia and age-related memory loss. Publishing in the journal Aging Cell, scientists were able to show, using experiments on mice that normally develop Alzheimer's symptoms less than a year after birth, that a daily dose of fisetin prevents the progressive memory and learning impairments commonly associated with the disease.
Fisetin from fruits and vegetables calms brain inflammation to prevent Alzheimer's symptoms
Senior staff scientist Dr. Pamela Maher commented, "We had already shown that in normal animals, fisetin can improve memory. What we showed here is that it also can have an effect on animals prone to Alzheimer's."
Interestingly, the researchers determined that fisetin did not alter the formation of amyloid plaques in the brain, accumulations of proteins which are commonly blamed for Alzheimer's disease. The new finding suggests a way to treat Alzheimer's symptoms independent of targeting amyloid plaques.
The study team found that, in the brains of mice genetically modified to develop Alzheimer's, pathways associated with inflammation at the cellular level were activated. But in the brains of fisetin-treated mice, the scientists identified anti-inflammatory molecules that quieted the inflammation. In particular, scientists determined that fisetin affected a protein known as p35 that has been implicated in turning on inflammatory pathways. Dr. Maher noted, "What we realized is that fisetin has a number of properties that we thought might be beneficial when it comes to Alzheimer's. ... Even as the disease would have been progressing, the fisetin was able to continue preventing symptoms."
It is important to note that fisetin was found to help prevent Alzheimer's progression, and no determination was made that the compound can reverse the disease after it has initiated. As with many chronic disease processes, lifestyle modification is a critical component to prevention. Dr. Maher concluded, "It may be that compounds like this that have more than one target are most effective at treating Alzheimer's disease, because it's a complex disease where there are a lot of things going wrong."
Regular consumption of strawberries and cucumbers and other natural fruits and vegetables can provide a sufficient dose of fisetin to help fight inflammation-mediated diseases including Alzheimer's dementia.
Sources for this article include:
http://onlinelibrary.wiley.com
http://www.sciencedaily.com
http://science.naturalnews.com
Suzanne
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Re: Alzheimer's Disease
[Re: Suzanne]
#166852
07/15/14 11:24 PM
07/15/14 11:24 PM
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Smell the Peanut Butter
What it Detects: Alzheimer's Disease.
How it Works: Alzheimer's typically affects our sense of smell because the area of the brain that processes odors, the olfactory cortex, is the first to show signs of dysfunction. (Memory problems come later.) That's why a researcher at the University of Florida, developed the peanut butter test, the results of which are published in the Journal of the Neurological Sciences.
Normal aging can affect our sense of smell, but peanut butter isn't an odor usually lost with time, according to researchers, making it a good tool for evaluating early Alzheimer's. In the study, they measured the distance at which participants could smell peanut butter through the left nostril compared with the right. Those with early Alzheimer's could not detect the smell until it was an average of almost 5 inches to the left nostril compared with the right.
While it is difficult to perform the test by yourself, you will need a partner. Close your eyes ask your partner to held a small jar of peanut butter 12 inches away from your left nostril while you hold your right nostril closed. Slowly move the jar closer until you are able to detect the smell. Now test your right nostril. You should be able to smell the peanut butter equally well in both nostril. If you can't, see your doctor to rule out treatable conditions that affect smell. AARP The Magazine
Suzanne
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Re: Alzheimer's Disease
[Re: Suzanne]
#169979
11/19/14 12:52 AM
11/19/14 12:52 AM
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Dietary and Lifestyle Changes Can Reverse Memory Loss In Alzheimer's, Study Proves
by PF Louis
(NaturalNews) A recent clinical trial was jointly conducted by the UCLA Mary S. Easton Center for Alzheimer's Disease Research, located in Los Angeles, CA, and the Buck Institute for Research on Aging in Novato, CA, 30 miles north of San Francisco. Those two combined resources to try a new approach for Alzheimer's patients that combined several protocols according to individual needs.
Their purpose was to prove that dietary and lifestyle changes could reverse or reduce memory and cognitive dysfunction, even Alzheimer's, with minimal pharmaceutical intervention. The researchers tried bundles of mostly dietary changes, supplements and exercise routines on 10 trial participants who were suffering with various stages of Alzheimer's disease (AD) and other symptoms of dementia.
Since first being identified some 100 years ago, cases of Alzheimer's have risen exponentially to become the sixth leading cause of death. At least that's according to the CDC. But one wonders if there is a blurred distinction between cause of death from Alzheimer's or simply death with Alzheimer's, as pneumonia is usually involved.
Even if one physically survives long after Alzheimer's has become so advanced that one no longer knows one's family or has any memory of his or her personal history or can barely function and often functions inappropriately, that person has become a total invalid. The prevailing actual medical causes of Alzheimer's patients is pneumonia.
After 100 years, mainstream medicine has no safe pharmaceutical solutions for Alzheimer's victims, who number over 5 million in the USA. The FDA has approved five drugs for treating Alzheimer's, but not one drug has been successful at even slowing AD's progress. The usual therapeutic approach for Alzheimer's has been combining several drugs simultaneously, which creates a cascade of side effects without curing the AD.
The trial used a flexible 36-point therapeutic system
The trial's report was published in the online journal Aging. Dale Bredesen, professor of neurology and director of the Easton Center at UCLA, developed the 36-point therapy that was tested in this clinical trial.
He explained how each patient needed to have the program suited to his or her special needs, general health and physiology. But usually the maximum pharmaceutical use was one drug only that could be reduced to none as long as the FDA, AMA and Big Pharma aren't looking. Perhaps that minimal drug use was to stay within "standard of care" guidelines and avoid the wrath of the AMA.
The results were more favorable than any pharmaceutical trial study, and no one died. Only one patient didn't improve. That was a person with advanced Alzheimer's. A few who were no longer able to work returned to work fully functional.
The problem with this approach within the medical monopoly prescription-writing one-size-fits-all mindset is the fact that it's complex and needs to be personalized and monitored closely. Dr. Bredesen can't clone himself.
Coconut oil
Coconut oil is legal everywhere, and it's cheap. Coconut oil's efficacy for even advanced Alzheimer's was discovered by Dr. Mary Newport when she missed out on getting her husband into the latest drug trial for Alzheimer's drugs.
Mary realized that the crux of the new synthetic drug dealt with medium chain triglycerides (MCTs). MCTs are easily converted by the liver into ketones, which can be used by a brain struggling with oxygen for energy.
She discovered that coconut oil contained high amounts of MCTs and began feeding her husband 2 to 4 tablespoons daily. His late-stage Alzheimer's soon improved considerably.
http://www.alz.org [PDF]
http://www.newswise.com
http://www.naturalnews.com
http://www.naturalnews.com
http://science.naturalnews.com
Suzanne
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Re: Alzheimer's Disease
[Re: Suzanne]
#171662
02/04/15 07:45 PM
02/04/15 07:45 PM
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New study shows these common over-the-counter drugs linked to Alzheimer's and other forms of dementia
by Kali Sinclair
(NaturalNews) A new study clearly shows common over-the-counter medications widely used for asthma, allergies, and insomnia increase the risk for dementia. The study also found that the risk may not be reversible, even after several years off of these medications.
The drugs studied are anticholinergic drugs, which block a neurotransmitter, acetylcholine, in the central and the peripheral nervous system. These drugs are available over-the-counter and through prescriptions. Many are used on a regular, long-term basis to treat a variety of medical complaints including:
* allergies and allergic reactions
* motion sickness
* irritable bowel syndrome
* insomnia
* colds
* ulcers
* asthma
* excessive sweating
* COPD
* urinary incontinence
* depression
* smoking cessation
* cough suppressant
* muscle relaxer
* Parkinson's disease
These drugs include:
* Zyban
* Wellbutrin
* Dramamine
* Cogentin
* Chlor-Trimeton
* Benadryl
* Sominex
* Advil PM
* Unisom
* Robinul
* Atarax
* Atrovent
* Oxivent
The study showed the risk of dementia rises when these anticholinergic medications are used at higher doses or for longer periods. It is not unusual to find these drugs used in combination, especially with the elderly population. For instance, one individual may be using a sleep aid, an allergy medication, and a medication for depression. If all of these drugs are anticholinergic drugs, the accumulative effect increases exponentially, raising the risk of dementia.
Earlier studies had shown these medications increased the risk of dementia; however, these studies also concluded cognitive decline decreased when these drugs were discontinued. The new study, a more longitudinal study, suggests that years after continuation, the risk of dementia remains higher for people who have used more of these drugs or used them for a longer period of time, suggesting a cause and effect even when the drugs were discontinued years prior to the development of dementia.
The study showed that even low amounts over long periods of time increased the risk of dementia. While the study focused on older adults, what about children and young adults? How many children and younger adults are regularly taking anticholinergic over-the-counter or prescription medications for allergies or asthma? How many adults of all ages are taking anticholinergic over-the-counter or prescription sleep aids?
This should be a wake up call for all of us to remember that medications treat symptoms, not the cause of disease. To get well, to be truly well, we must give the body the nutrients it needs to heal. We must detox. We must live lives that are as clean as possible: clean food, clean water, clean environment. It has been made clear over and over that we cannot trust pharmaceutical companies! If you have been taking any pharmaceuticals, it's time to detox. Start with this Cheap and Easy Detox Plan and consider following the 80% Raw Food Diet.
Sources:
http://www.medscape.com
http://www.sciencedaily.com
The study: Shelly L. Gray, Melissa L. Anderson, Sascha Dublin, Joseph T. Hanlon, Rebecca Hubbard, Rod Walker, Onchee Yu, Paul K. Crane, Eric B. Larson.Cumulative Use of Strong Anticholinergics and Incident Dementia.JAMA Internal Medicine, 2015; DOI:10.1001/jamainternmed.2014.7663
Suzanne
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Here is the link to this week's Sabbath School Lesson Study and Discussion Material: Click Here
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